Antithrombin-III deficiency in ischemic stroke.
نویسندگان
چکیده
منابع مشابه
Case Report Cerebral Infarction in a Heterozygote With Variant Antithrombin III
Background: We report a heterozygous case of familial qualitative deficiency of antithrombin III associated with cerebral infarction. Case Description: A 33-year-old man had a history of recurrent transient ischemic attacks from the age of 28. Cerebral computed tomography at age 29 disclosed a low-density area in the left frontal lobe, and an internal carotid angiogram showed branch occlusion o...
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BACKGROUND AND PURPOSE One or more of the inherited thrombophilias may be causal risk factor for a proportion of ischemic strokes, but few studies have addressed this association or the association between thrombophilia and pathogenic subtypes of stroke. METHODS We conducted a case-control study of 219 hospital cases with a first-ever ischemic stroke and 205 randomly selected community contro...
متن کاملCerebral infarction in a heterozygote with variant antithrombin III.
BACKGROUND We report a heterozygous case of familial qualitative deficiency of antithrombin III associated with cerebral infarction. CASE DESCRIPTION A 33-year-old man had a history of recurrent transient ischemic attacks from the age of 28. Cerebral computed tomography at age 29 disclosed a low-density area in the left frontal lobe, and an internal carotid angiogram showed branch occlusion o...
متن کاملIschemic stroke due to deficiency of coagulation inhibitors. Report of 10 young adults.
BACKGROUND AND PURPOSE Deficiencies in coagulant inhibitors protein C, protein S, and antithrombin III increase the risk of venous thrombosis. We describe 10 young adults with cerebral arterial thrombosis due to deficiencies in these factors. METHODS Sixty patients younger than 45 years were hospitalized because of acute ischemic stroke diagnosed through computed tomography or magnetic resona...
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To evaluate the role the coagulation and fibrinolysis abnormalities in the pathogenesis of ischemic stroke of undetermined etiology, we assayed plasma concentration of fibrinopeptide-A and thrombin-antithrombin III complex, both sensitive markers for thrombin activation and fibrin formation, and D-dimer, a marker of plasmin activity and fibrinolysis. Hemostatic markers were measured in 32 patie...
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ورودعنوان ژورنال:
- Stroke
دوره 21 6 شماره
صفحات -
تاریخ انتشار 1990